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- Polycythaemia as a complication of transdermal testosterone therapy.
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- Direct myocardial cell damage could occur by disruption of mitochondria and induction of intrafibrillar collagen dysplasia (Melchert and Welder 1995; Sullivan et al 1998).
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If you realize and belief the website you’re making an attempt to entry, turn off the Threat Protection characteristic within the app settings and examine out again. If you can entry the site with Threat Protection offand you imagine that NordVPN is blocking a net site it should not be, please inform our customer support brokers. Effects of testosterone on coronary vasomotor regulation in men with coronary coronary heart disease.
Anabolic Steroids, Acute Myocardial Infarction And Polycythemia: A Case Report And Review Of The Literature
Erythrocytosis has been reported as a potential effect of testosterone, which could be helpful in patients with anemia. The greatest risk of erythrocytosis happens with injectable types of testosterone in contrast with transdermal preparations, especially in supraphysiologic doses. An enhance of as a lot as 44% of the baseline hematocrit has been reported .
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Rhoden EL, Morgentaler A. Risks of testosterone-replacement therapy and proposals for monitoring. Testosterone alternative in hypogonadal men with angina improves ischaemic threshold and quality of life. Ajayi AAL, Mathur R, Halushka PV. Testosterone enhance human platelet thromboxane A2 receptor density and aggregation responses.
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We report a case of an acute ST-segment elevation myocardial infarction in a patient with conventional cardiac risk elements using supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this patient also had polycythemia, doubtless secondary to high-dose testosterone. The patient underwent profitable percutaneous intervention of the proper coronary artery. Physicians and patients ought to concentrate on the clinical consequences of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).
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